The Role of Nitric oxide in IL-1β-mediated Dysfunction of Rodent Islets of Langerhans: Implications for the Function of Intrahepatic Islet Grafts
Document Type
Article
Publication Date
1994
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Abstract
Products of inflammation, such as interleukin-1β (IL-1β) and nitric oxide (NO), may impair early function of pancreatic islet grafts. In in vitro studies, freshly isolated rat islets of Langerhans cultured for 24 hr (10 islets/well) in the presence of 20 IU/ml of IL-1β released 57% less insulin (mean ± S.E. of 151±61 μU) on the average than control (385±89 μU) cultures (n=9, P=0.08). Nitrite levels in the medium (indirect measure of NO) after islets were cultured for a 24-hr period were nearly 3-fold greater in IL-1β-exposed islets than control islet cultures (5.8±1.0 μM vs. 2.2±0.3 μM, P=0.03). Production of nitrite by islet cells in the presence of IL-1β was inhibited in cultures also containing 2 mM L-NG-monomethyl-Arginine (L- NMMA) (3.4±0.4 μM, n=9, P=0.09 vs. control). When islets were maintained for 1 hr in 30 mg/dl glucose followed by 300 mg/dl for 1 hr, insulin release (stimulated) increased 6-fold (from 7±2 to 45±11 μU) in control cultures but only 3-fold (from 4±2 to 12±4 μU) in IL-1β-exposed cultures (n=9, P=.01). Addition of 2 mM L-NMMA to islet cultures in the presence of IL-1β (20 IU/ml) (n=9) restored insulin release to normal (from 6±2 to 38±9 μU, P≥0.6), suggesting that NO mediates the inhibitory effect of IL-1β on beta- cell function. In in vivo studies, rats with streptozotocin-induced diabetes (blood glucose >400 mg/dl) received minimal (750 hand-picked islets) intraportal beta cell mass isografts with (n=5) or without (n=9) treatment with 100 mg/7 days of L-NMMA from 3 days before transplantation to 4 days after transplantation (POD -3 to +4). L-NMMA-treated rats became euglycemic (glucose <200 mg/dl) earlier than nontreated rats (mean ± SD of 6.4±2.5 vs. 16.7±4.7 days posttransplant, P=0.001). These findings support the hypothesis that NO is a mediator of beta cell dysfunction after intraportal transplantation of freshly isolated islets of Langerhans.
Repository Citation
Xenos, E. S.,
Stevens, R. B.,
Sutherland, D. E.,
Lokeh, A.,
Ansite, J. D.,
Casanova, D.,
Gores, P. F.,
& Platt, J. L.
(1994). The Role of Nitric oxide in IL-1β-mediated Dysfunction of Rodent Islets of Langerhans: Implications for the Function of Intrahepatic Islet Grafts. Transplantation, 47 (8), 1208-1212.
https://corescholar.libraries.wright.edu/surg/403